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Interestingly, MOTS-c improve the survival status of mice during methicillin-resistant staphylococcus aureus infection and promote the AhR/Stat3 signaling pathway by declining pro-inflammatory cytokines (TNF-α, IL-6, and IL-1β) and increasing anti-inflammatory cytokines IL-10 [51]
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XPA‐deficient cells showed defective mitophagy with excessive cleavage of PINK1 and increased mitochondrial membrane potential
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